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Dylan Morris
Dylan Morris

Next Level Metabolism By Jade Teta.epub !!TOP!!

Earlier, I talked about how your thyroid must produce the right amounts of hormones to support your metabolic rate. Insulin, a hormone that regulates blood sugar levels, is another key player in metabolism.

Next Level Metabolism by Jade Teta.epub

What you eat and how you move can significantly impact your metabolism. Focusing on sufficient protein during all your meals, weight resistance, HIIT, and optimizing your thyroid and blood sugar levels will optimize your metabolic rate.

The two most important appetite regulating hormones are leptin and ghrelin and exercise impacts both (2-6). Leptin is produced in the fat cell and acts like the body's fuel gauge communicating to the brain how much fuel (fat) it has in its tank (adipose tissue). Rising leptin levels shut off hunger and stimulate metabolism via thyroid and adrenal stimulation. Falling leptin levels induce appetite and slow metabolism.

One confusing addendum to this mechanism is that when leptin levels are very high for long periods of time, leptin resistance sets in. This means both very high and low levels of leptin can cause increased hunger and slowed metabolism. Leptin is usually thought of as a day-to-day appetite regulator.

If for those that know a bit about hormone metabolism, if I sort of start at the beginning. For those that don't, you'll still be interested or you could always listen to this again and pull out a picture or Google hormone or oestrogen metabolism and then sort of follow through. But the first one I would mention would be the CYP or the CYP17A1. CYP17A1 is something that converts progesterone or pregnenolone into the androgens. And there's a genetic variation that can increase enzyme activity. So that would mean that progesterone could get pushed over to the androgens. Therefore that person might be at risk of having a little lower oestrogen levels, more of the androgens.

CYP1B1, CYP1A1, both of these are actually involved in not just hormone metabolism but also breaking down or metabolising what are known as PAHs, or polycyclic aromatic hydrocarbons. These are things found in combustion fuels, so think about car exhaust fumes, also cigarette smoke. Even not they're the main players but even a bit of breaking down caffeine. It's really CYP1A2 that's the main player in caffeine. But if you are consuming all of these things or exposed to a lot of these chemicals, that can also influence the metabolism of these oestrogens. So we can test for these SNPs and say to someone, "Well, you are more at risk of having this 4-hydroxy estrogen. That could be part of the reason why you've got higher levels of this particular metabolite." Or, if they haven't had testing done, it might then trigger you to say, "Well, you do have high oestrogen. You have a genetic variation that makes you more susceptible to the 4-hydroxy. We want to test this and find out do you actually have high levels, because if you do, we want to start to treat that or try to manage that as quick as possible." Because the 4-hydroxy as well as the 16 but particularly the 4-hydroxy can cause the quinones which leads to DNA damage and then, you know, DNA damage as we know can cause a number of outcomes including cancers. 041b061a72


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